Researchers at the University of Michigan School of Medicine have made a correction to a previously published study on cancer metabolism and tumour immunology. The correction, published in the journal Nature, relates to a figure in the original article that was mistakenly duplicated.
According to the correction, the representative flow cytometry dot plot for condition SupSer in Extended Data Fig. 1j was mistakenly a duplicate image of condition A375 sup. The figure has been updated in both the HTML and PDF versions of the article. The correction was made to ensure the accuracy and reliability of the study's findings.
The original study, published on September 2, 2020, explored the role of the SLC43A2 gene in cancer metabolism and its impact on T cell methionine metabolism and histone methylation. The research team, led by Yingjie Bian and Wei Li, discovered that the SLC43A2 gene plays a crucial role in regulating T cell function and that its alteration can lead to changes in methionine metabolism and histone methylation.
"We are committed to ensuring the accuracy and reliability of our research findings," said Dr. Weiping Zou, a co-author of the study and director of the University of Michigan Rogel Cancer Center. "We appreciate the opportunity to correct this mistake and ensure that our research contributes to the advancement of cancer treatment and understanding."
The correction is a testament to the scientific community's commitment to transparency and accuracy. The study's findings have significant implications for our understanding of cancer metabolism and tumour immunology, and the correction ensures that the research remains reliable and trustworthy.
The University of Michigan School of Medicine continues to be at the forefront of cancer research, with a focus on understanding the complex interactions between cancer cells and the immune system. The correction to the study is a minor setback, but it highlights the importance of rigorous scientific inquiry and the need for ongoing research to advance our understanding of cancer and its treatment.
The study's findings have sparked interest in the scientific community, and researchers are continuing to explore the role of the SLC43A2 gene in cancer metabolism and tumour immunology. The correction is a minor development in an ongoing effort to understand and combat cancer, and it serves as a reminder of the importance of accuracy and transparency in scientific research.
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